developing allergy imprints. This ability seems partially due to a certain genetic predisposition.
Once formed, the allergy imprint remains in place. It is inactive unless it comes into contact with the allergen. Activated by the specific information that characterizes its imprint, it triggers the allergic reaction via impulses using the known mechanisms, i. e. antibody formation, mast cell sensitization, release of histamine, etc.
Typical for this kind of “allergy imprint” is not only the specificity—it is unique to a particular range of frequency information—but also the ability to manipulate it. Moreover, it can be treated with biophysical means. That is to say, by applying suitable information or techniques it can be attenuated and/or completely eliminated.
None other than the allergen's biophysical frequency pattern (= physics codification) is the “suitable information” for the “allergy imprint.” Employing this specific signal and suitable implementation, it is possible to “register” this imprint, that is, to determine its presence, for example by means of the allergen resonance test and/or influence it therapeutically.
The ideas outlined here cannot (yet) be verified with data and do not claim correctness according to the current scientific point of view. They have, however, proven useful as a thought modality as they elucidate all phenomena occurring while treating a patient.
For biochemical immunological processes to take place on the material level, contact with an allergen is necessary to activate the allergy imprint.
Using the known mechanism, these biochemical immunological processes now initiate an allergic reaction. The patient shows symptoms such as irritated skin and mucous membrane, itching and edema, etc.
When the patient is episodically exposed to the allergen (“acute allergy” model, see Classification of Allergies, p. 49), the allergic reaction will subside. It will subsequently reappear upon renewed contact with the allergen.
Being continuously or frequently exposed to the allergen will result in chronic allergic stress that may eventually influence the entire organism. The allergic reaction turns into an allergic disease that can become increasingly independent, with the connection to the allergen becoming less and less obvious (e. g. neurodermatitis, chronic bronchial asthma). We will describe these interrelations in more detail in Chronic Forms of Allergies, page 50.
Fig. 3.3 Simplified thought modality to explain physical allergy mechanisms (see text).
The schematic illustration in Figure 3.3 is meant to once again demonstrate the hierarchy of the “levels.” It shows that in all cases the informational level needs to emit an impulse for corresponding reactions to take place on the material level.
4 Symptoms of Allergic Reactions
Allergic reactions can manifest in so many ways throughout the body that we need to consider an allergy when looking at almost every medical symptom!
The most common symptoms are listed in the table below:
Most Common Symptoms of Allergic Reactions
General symptoms:
Fatigue, decreased performance, feeling cold, vertigo
Cutaneous symptoms:
Exanthema (maculate, popular, urticarial, etc.)
Quincke's edema, itching, neurodermatitis—all severities
Irritation of the mucous membrane:
Rhinitis, sneeze impulse, conjunctivitis, itchy eyes, cough, bronchial asthma
Gastrointestinal symptoms:
Feeling of repletion, gastritis, stomatitis, diarrhea, meteorism, Roemheld's syndrome, colitis in various forms
Heart and circulatory symptoms:
Variations in blood pressure, syncope, tachycardia, extrasystole
Bladder symptoms:
Polyuria, dysuria, irritable bladder, susceptibility to urinary tract infections
Muscles, joints:
Sore muscles, rheumatic pain
Psychological symptoms:
Depression, restlessness, confusion, feeling dazed, anxiety and panic attacks, aggressiveness, hyperactivity in children
Headaches and migraines
Weight loss and gain
Anaphylactic shock
The location, intensity, and time of appearance of allergic symptoms depend on various factors:
Allergy symptomatology depends on:
Usually the prerequisite is a genetic predisposition. The ability to create an allergy is inherited, not the type of allergy or the substance that causes it.
The allergen potency of a trigger substance varies. Aggressive allergens are substances that easily cause sensitivities (e. g., grass pollen, cat hair, strawberries). Therefore the clinical symptomatology caused by these substances occurs frequently.
Allergic reactions vary depending on the general state of the organism.
Any somatic stress (other illnesses, focal infections, toxic or geopathic stressors) can intensify a reaction.
Particularly important is any kind of psychological stress. Fear, grief, excitement, being overtaxed at work or school, etc. can cause the onset of or intensify allergic symptoms. On the contrary, these symptoms may improve considerably or disappear completely in a relaxed, harmonious situation.
Most people with allergies have particular “target organs” that tend to manifest allergic reactions (specifically skin, bronchials, intestinal mucosa).
Most textbooks used in clinical allergology are based on the type classification established by Coombs and Gell in 1963 (Fig. 5.1).
Fig. 5.1 Immunological mechanisms of the four most important allergy types. Illustration according to Roitt, Brost-off, and Male: Kurzes Lehrbuch der Immunologie (Short Treatise of Immunology), 1987.